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TLR4 activation in platelets whether by pathogen- (viremia) or damage-associated molecular patterns induces a prothrombotic and proinflammatory state (Schattner, 2019)

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The first generation monoclonal antibodies against Aβ monomer did not show a meaningful side in clinical trials, while the second generation monoclonal antibodies (such as aducanumab, lecanemab, donanemab) recently approved for clinical research clearly proved that targeting the Aβ pathway can delay the progression of early AD by directly clearing Aβ plaque in the brain (Kim et al., 2025)