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Paracetamol/acetaminophen hepatotoxicity: new markers for monitoring the elimination of the reactive N-Acetyl-p-benzoquinone imine | bioRxiv

This concentration should not result in significant cell death in cultured hepatocytes, regardless of the clinical Cmax differences of the drugs ( The toxic event, which progresses from molecular initiating events to secondary events and ultimately results in severe cell dysfunction and death, can be best described as a sequential process occurring over time

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The clinical evidence-based analysis has found that oral supplementation is as effective as intramuscular injections